Scientists now admit that COVID-19 can change a father’s sperm and affect his children’s mental health. But here’s what they won’t say out loud: could the real culprit be the same spike protein mechanism triggered by the shots themselves?
Both COVID-19 and the mRNA shots interact with the body through the ACE2 receptor and the spike protein. If that process can alter sperm and brain development in offspring, as the study suggests, it raises serious questions about what’s really behind these intergenerational changes—and whether “the virus” is being used as a convenient scapegoat.
Researchers at the Florey Institute of Neuroscience and Mental Health found that when male mice were infected with SARS-CoV-2, their sperm underwent measurable molecular changes that affected the next generation. Offspring—especially females—showed signs of increased anxiety and stress sensitivity, despite never being exposed to the virus themselves.
“We already knew that when male mice were exposed to specific environmental and lifestyle factors, like poor diet before mating, it could change brain development and behavior in offspring,” explained Professor Anthony Hannan, head of The Florey’s Epigenetics and Neural Plasticity Research Group.
“This is because the father’s experiences can alter the information carried in sperm, including specific RNA molecules, which transmit instructions for offspring development.”
The researchers infected genetically modified mice that carry the human ACE2 receptor—allowing the virus to invade cells much like it does in humans. Infection caused testicular inflammation, reduced sperm count and motility, and damage to sperm-producing cells, while immune cells invaded the testes and inflammatory genes were switched on.
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| Covid lockdowns have been linked to permanent neurological impairment in children; now Covid infections and vaccines have also been linked to long term mental health issues in children who were not even born during the pandemic |
When these males were bred with healthy, uninfected females, their offspring showed anxiety-like behaviors and heightened stress reactions. The researchers confirmed that these differences weren’t caused by infection in the young, but by changes inherited through the father’s sperm.
Further molecular analysis revealed that SARS-CoV-2 infection altered small noncoding RNA molecules (sncRNAs) in sperm—epigenetic markers that can switch genes on or off during early development. These molecular “scars” didn’t change the DNA sequence, but they influenced how the offspring’s genes were expressed, particularly in the hippocampus—the brain region tied to emotion, mood, and anxiety.
“These kinds of changes in the hippocampus, as well as other brain regions, may contribute to the increased anxiety we observed in offspring, via epigenetic inheritance and altered brain development,” said Dr. Carolina Gubert, co-author of the study.
While the researchers emphasized that their work was done in mice, not humans, the implications are sobering. “If our findings translate to humans, this could impact millions of children worldwide, and their families, with major implications for public health,” said Hannan.
Mainstream outlets have framed the study as a warning about “the long shadow” of COVID infection—but avoided the elephant in the room: the same spike protein, the same receptor, the same inflammatory pathways are activated by mRNA injections. Yet no equivalent studies have examined whether these so-called vaccines might also leave similar molecular traces in sperm.
Could the establishment be blaming the virus to avoid opening that door? If the damage is immune-mediated or spike-related rather than infection-specific, then much of what’s being labeled as a “post-viral effect” could, in fact, be something else entirely.
Either way, the science from The Florey Institute makes one thing clear: what happens at the molecular level during these exposures doesn’t just affect one generation. It may echo far into the next.